New Danger From Taking Vitamin D Supplements
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New Danger From Taking Vitamin D Supplements

By Robert Cohen,

Most women have never heard of milk-alkali syndrome, and that omission of USDA and FDA marketing is criminal. Federal agencies continue to promote
milk consumption without offering the dangerous facts regarding milk consumption.

Before getting to a new study published in the September, 2010 issue of the Journal of the American Society of Nephrology (Sep;21(9):1440-3) let me
shed some light on the milk-alkali syndrome.

Got excessive urination and back pain?
Got nausea and/or fatigue?
Got kidney stones?

All of the above are symptoms of milk-alkali syndrome.

Milk-alkali syndrome is caused by consuming cow's milk which can cause gastric reflux, and certain antacids used to counter the painful effects of that same reflux caused by...yep--milk consumption.

The components of antacids neutrilize gastric pH and relieve the sufferer from discomfort, but these ingredients particularly sodium bicarbonate
and calcium bicarbonate result in large calcium deposits in the kidneys.

Over time, these calcium deposits result in the rarely diagnosed condition of milk-alkali syndrome.

And...guess what makes this condition much worse? Guess what accelerates the probability of an emergency room visit?

Vitamin D supplements and Vitamin D added to milk.

Now for the abstract of the study every milk-drinking woman should read:

Got calcium? Welcome to the calcium-alkali syndrome.
Patel AM, Goldfarb S.

Renal-Electrolyte and Hypertension Division, Department of Medicine, University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania 19104, USA.


"We recommend changing the name of the milk-alkali syndrome to the calcium-alkali syndrome, because the new terminology better reflects the shifting epidemiology and understanding of this disorder. The calcium-alkali syndrome is now the third most common cause of hospital admission for hypercalcemia, and those at greatest risk are postmenopausal or pregnant women.

"The incidence of the calcium-alkali syndrome is growing in large part as a result of the widespread use of over-the-counter calcium and vitamin D supplements. Advertising for treatment or prevention of osteoporosis has long encouraged this use. Intricate mechanisms mediating the calcium-alkali syndrome depend on interplay among intestine, kidney, and bone. New insights regarding its pathogenesis focus on the key role of calcium-sensing receptors and TRPV5 channels in the modulation of renal calcium excretion. Restoring extracellular blood volume, increasing GFR and calcium excretion, and discontinuing calcium supplementation provide best treatment."

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